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OCR: sult of Apo B levels can also be increased as a at consists excessive levels of Lp(a), a lipoprotein Fig. 6.16). of apo (a) in disulfide linkage with LDL .p(a) are The source, fate, and precise function o erminant not yet known, although the primary d (a) has of Lp(a) levels appears to be genetic. Ap considerable structural homology with ysis; high plasminogen. Lp(a) may therefore play physiological role in modulating fibrin Ik of levels are associated with an increased erosc ler